日本消化器病学会アーカイブシステム

セッション	検索結果は4件です。 TAK1 is a Gatekeeper Protein that Prevents Spontaneous Apoptosis, Inflammation, Fibrosis and Carcinogenesis in the Liver Seki　Ekihiro（Division of Gastroenterology, Department of Medicine, University of California, San Diego, School of Medicine, USA） Transforming growth factor β-activated kinase 1（TAK1）is a MAP kinase kinase kinase that regulates activation of NF-κB and JNK pathways in TLRs, IL-1, TNF and TGFβ signaling. NF-κB and JNK are essentia... 第99回日本消化器病学会総会＞ 1）Inflammation and Hepatocellular Carcinoma Growth Factors in Liver Regeneration and Hepatocarcinogenesis：New Mechanisms and Interactions of the EGFR Signalling System Avila　Matias A（Division of Hepatology and Gene Therapy, CIMA, University of Navarra and CIBERehd, Spain） Hepatocellular carcinoma（HCC）slowly unfolds on a background of chronic liver injury, fibrosis and inflammation. Interestingly, a number of key mechanisms that participate in the normal liver regenerat... 第99回日本消化器病学会総会＞ 1）Inflammation and Hepatocellular Carcinoma Reversible Phospho-Smad Signaling between Tumor-suppression and Fibro-carcinogenesis in Human Chronic Liver Diseases Matsuzaki　Koichi（Department of Gastroenterology and Hepatology, Kansai Medical University, Japan） Clinical observations suggest that chronic inflammation associated with hepatitis B or C virus（HBV or HCV）infection simultaneously induces hepatic fibrosis and carcinogenesis（fibro-carcinogenesis）. Co... 第99回日本消化器病学会総会＞ 1）Inflammation and Hepatocellular Carcinoma Cancer Stem Cells in Hepatocarcinogenesis Yamashita　Taro（Department of Gastroenterology, Kanazawa University Graduate School of Medical Science, Japan） Carcinogenesis could be characterized as deregulated malignant organogenesis mediated by abnormally proliferating and/or metastatic cancer cells and activated stromal cells that trigger angiogenesis, ... 第99回日本消化器病学会総会＞ 1）Inflammation and Hepatocellular Carcinoma

セッション

検索結果は4件です。

TAK1 is a Gatekeeper Protein that Prevents Spontaneous Apoptosis, Inflammation, Fibrosis and Carcinogenesis in the Liver: Seki　Ekihiro（Division of Gastroenterology, Department of Medicine, University of California, San Diego, School of Medicine, USA）
Transforming growth factor β-activated kinase 1（TAK1）is a MAP kinase kinase kinase that regulates activation of NF-κB and JNK pathways in TLRs, IL-1, TNF and TGFβ signaling. NF-κB and JNK are essentia...
第99回日本消化器病学会総会＞ 1）Inflammation and Hepatocellular Carcinoma
Growth Factors in Liver Regeneration and Hepatocarcinogenesis：New Mechanisms and Interactions of the EGFR Signalling System: Avila　Matias A（Division of Hepatology and Gene Therapy, CIMA, University of Navarra and CIBERehd, Spain）
Hepatocellular carcinoma（HCC）slowly unfolds on a background of chronic liver injury, fibrosis and inflammation. Interestingly, a number of key mechanisms that participate in the normal liver regenerat...
第99回日本消化器病学会総会＞ 1）Inflammation and Hepatocellular Carcinoma
Reversible Phospho-Smad Signaling between Tumor-suppression and Fibro-carcinogenesis in Human Chronic Liver Diseases: Matsuzaki　Koichi（Department of Gastroenterology and Hepatology, Kansai Medical University, Japan）
Clinical observations suggest that chronic inflammation associated with hepatitis B or C virus（HBV or HCV）infection simultaneously induces hepatic fibrosis and carcinogenesis（fibro-carcinogenesis）. Co...
第99回日本消化器病学会総会＞ 1）Inflammation and Hepatocellular Carcinoma
Cancer Stem Cells in Hepatocarcinogenesis: Yamashita　Taro（Department of Gastroenterology, Kanazawa University Graduate School of Medical Science, Japan）
Carcinogenesis could be characterized as deregulated malignant organogenesis mediated by abnormally proliferating and/or metastatic cancer cells and activated stromal cells that trigger angiogenesis, ...
第99回日本消化器病学会総会＞ 1）Inflammation and Hepatocellular Carcinoma